Similar to the second study, reductions of oxidative and inflammatory responses were the primary findings, thereby demonstrating a potential acute recovery-enhancing effect between bouts of high-intensity aerobic exercise with tart cherry supplementation. A third endurance study examined the effects of 7-d tart cherry concentrate supplementation on physiological markers of muscle damage, oxidative stress, and inflammation surrounding 3-d of simulated high-intensity road cycling. Following a similar 8-d tart cherry juice supplementation protocol, a second study reported greater lower body isometric strength and quicker restoration of muscular function with reduced blood markers of muscle damage, oxidative stress, and inflammation in response to a marathon run. Exercise-induced muscle pain was reduced as a result of tart cherry supplementation, but the findings were not confirmed by subsequent blood marker analysis. The first endurance-based study investigated the effects of 8-d tart cherry cultivar-blended juice supplementation on exercise-induced muscle pain surrounding an endurance relay race event (running distance = 22.5–31.4 km). There are a few studies that have evaluated the effects of tart cherry supplementation on responses to endurance-based exercise. Mortmorency) cherry concentrate and juice supplementation to help increase performance by theoretically attenuating muscle damage, oxidative stress, and inflammation associated with aerobic challenges. Exercise-based research with similar functional foods spurred investigation with tart (e.g. A wide variety of antioxidant and polyphenol-containing functional foods such as grape extract, chokeberries, and blueberries have shown performance-enhancing and exercise recovery benefits. It is proposed that these may act synergistically with other compounds contained within the food to provide an overall aerobic exercise recovery benefit. More recent nutritional research has focused on the antioxidant effects of functional foods containing high concentrations of phenolic compounds such as flavonoids and anthocyanins. However, vitamins C and E (independently or in combination with N-acetylcysteine, β-carotene, or α-lipoic acid) remain controversial due to conflicting reports of effectiveness with potential post-exercise pro-oxidant effects on muscle protein anabolism, endogenous antioxidant capacity, and mitochondrial biogenesis. The use of antioxidant supplements, such as vitamins C and E, in athletic applications to help fortify the body’s endogenous antioxidant response has spurred some success. Exercise-induced muscle soreness is indirectly related to inflammation as a product of high nociceptor and mechanoreceptor sensitivity to potent metabolites released during muscular degeneration. Ultimately, this increase facilitates excessive cell damage, altered cell signaling, decreased cellular performance, lipid peroxidation, oxidation of proteins and glutathione, and subsequent DNA damage. As a result, this type of long duration mechanical muscle stress and high oxidative metabolic demand, significantly increases free radical production beyond the capacity of the endogenous antioxidant systems. Over the 48-h recovery period, P changes in medial quadriceps soreness from pre-run measures were smaller compared to TC.Īcute bouts of strenuous aerobic exercise facilitate a stress response characterized by mechanical muscle damage, oxidative stress, and inflammation that parallels the physiological stress response associated with many adverse traumatic cardiovascular events and illnesses. Soreness perception between the groups was different over time in the medial quadriceps ( p = 0.035) with 34 % lower pre-run soreness in TC compared to P. Inflammatory markers were 47 % lower in TC compared to P over time ( p = 0.053) coupled with a significant difference between groups ( p = 0.017). Despite lower antioxidant activity pre-run in TC compared to P, changes from pre-run levels revealed a linear increase in antioxidant activity at 24 and 48-h of recovery in TC that was statistically different (16–39 %) from P and pre-run levels. Attenuations in TC muscle catabolic markers were reported over time for creatinine ( p = 0.047), urea/blood urea nitrogen ( p = 0.048), total protein ( p = 0.081), and cortisol ( p = 0.016) compared to P. Subjects in the TC group averaged 13 % faster half-marathon race finish times ( p = 0.001) and tended to have smaller deviations from predicted race pace ( p = 0.091) compared to P.
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